Primary hyperoxaluria: A case report


Case Report

Author Details : Ankita Deshmukh*, Shweta Nemade, Ramesh Kalshetty, Anitha Padmanabhan, Nitin Gadgil

Volume : 10, Issue : 2, Year : 2023

Article Page : 195-197

https://doi.org/10.18231/j.ijpo.2023.041



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Abstract

Renal failure can occasionally be caused by oxalate nephropathy. Glyoxylate metabolism abnormalities and particular hepatic enzyme deficits are the causes of primary hyperoxaluria (PH). Increased intestinal absorption, an excessive diet or an increased intake of oxalate precursors can all result in secondary hyperoxaluria. A 13-month old male child with high blood creatinine, low sodium, low calcium levels, high uric acid and low urine specific gravity is the subject of this research. Medullary papillary calcification was detected using sonography (nephrocalcinosis). Calcium oxalate crystals, sparse lymphocytic infiltration and interstitial fibrosis were seen on a renal biopsy. The patient was put on peritoneal dialysis, progresses to anuria and expired due to renal failure.
 

Keywords: Calcium oxalate, Crystal, Nephropathy, Primary hyperoxaluria, Renal failure.


How to cite : Deshmukh A, Nemade S, Kalshetty R, Padmanabhan A, Gadgil N, Primary hyperoxaluria: A case report. Indian J Pathol Oncol 2023;10(2):195-197


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Article History

Received : 06-04-2023

Accepted : 29-04-2023


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https://doi.org/ 10.18231/j.ijpo.2023.041


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